Diabetes:
Causes, Natural Control, & the Mercury/Vaccine Factor B. Windham(Ed)
2011
I. Introduction
Diabetes
(hyperglycemia) incidence is increasing drastically. For individuals born in
2000, the lifetime risk of diabetes in the U.S. is 33% and over 16 million in
the U.S. currently have diabetes(502d,580,598), while over 54 million have
insulin resistance or pre-diabetes(1). Childhood
diabetes is increasing even more rapidly than adult diabetes(82,493,502,598,1).
Over the last 20 years the percent of diabetes cases below 20 years old has
increased from 2% to over 30%, and there was a 70% increase in cases under 40
years of age between 1990 and 1998(502). Studies in the U.S. and Europe have
found toxic metals and vaccinations to be factors in the increased diabetes
cases (502,369,35,100,490-492) along with poor dietary habits(1).
A greatly increased incidence of juvenile diabetes has been correlated to
specific vaccination sequences and to the number of vaccines given (11,502).
Autoimmune diseases with a predominant Th1 cytokine component involvement in some cases include
insulin-dependent diabetes mellitus(12). Incidence of
insulin resistance which a factor in many chronic conditions has also increased
significantly. Diabetes and pre-diabetes are conditions rather than diseases,
and usually can be prevented or controlled – see Section V.
There are
2 basic types of diabetes, Type I (autoimmune) diabetes and Type II (insulin
resistance). In type I diabetes the body’s immune system attacks its own
insulin producing tissue, the beta cells of the pancreas (580). Type II
diabetes involves metabolic failure related to poor diet, obesity,
environmental factors, and genetic susceptibility (1). Insulin resistance is a
primary factor in type 2 diabetes and results when the body cannot properly use
insulin, which is secreted by the pancreas to move glucose from the blood into
the cells that need it. The pancreas then produces extra insulin in a futile
effort to compensate, leading to higher insulin and glucose levels in the blood
along with a deficiency of glucose in the cells that need sugar to function
properly. Those with either type I or type II diabetes have been found to be
more likely to have other chronic conditions including heart disease, strokes,
kidney disease, Alzheimer’s, eye conditions and blindness (580,581)
II. Causes of
Diabetes and the mercury/toxic metal connection
Type 2 diabetes and
pre-diabetes is an epidemic among young Americans and greatly increases the
incidence of heart attack, blindness, stoke, infertility, and early death.
Brain inflammation has been found to be a major factor in pre-diabetes,
diabetes, and the closely related metabolic syndrome which includes diabetes or
pre-diabetes, high blood pressure, and high cholesterol (598,1, 581b,etc.). Causes of oxidative stress and lipid peroxidative
related brain inflammation that have been documented include vaccines, mercury,
aluminum, excitotoxins such as MSG, aspartame, food additives, and
overconsumption of high-fructose corn sweetener, starches, sweets, and other
omega 6 oils (13,424,489,494,496,596-599,1,etc.).
These cause high glutamate levels in the brain and oxidative damage –resulting
in inflammation of the brain and immune system, as well as damage to brain
microglia cells and the mitochondrial DNA, fatigue, high triglycerides,
metabolic syndrome, etc. These have been found to be factors in most chronic
neurological diseases. Inflammation induced by vaccine adjuvants like aluminum
and mercury or by excitotoxins like MSG has been found to play a significant
role in insulin resistance (type-2 diabetes) and in high levels of LDL
cholesterol (597,598,etc.). There is
also evidence that the diet drink sweetener aspartame can cause or increase the
effects of diabetes and hypoglycemia (450,498). Higher levels of vit D have been found to be associated with lower levels of
insulin resistance and appears to be protective(580). Theoflavins from black or green tea and curcumin have been
found to be significantly effective in protection against inflammation and
inflammatory conditions(580).
Reduced
levels of magnesium and zinc are related to metabolic syndrome, insulin
resistance, and brain inflammation, and are protective against these conditions
(599,43). Mercury and cadmium inhibiting magnesium and zinc levels as well as
inhibiting glucose transfer are other mechanisms by which mercury and toxic
metals are factors in metabolic syndrome and insulin resistance/diabetes
(43,198,338,505, 597). Iron overload has also been found to be a cause of
insulin resistance/type 2 diabetes(595). Mercury also
has synergistic
effects with other toxics such as dioxins that have been found
to increase insulin resistance more than simply additive effects(505).
Mercury
exposure levels from vaccinations have likewise shown major increases during
this period and have been found to be a major factor in the increase of other
conditions like autism and ADHD(8). The most common
mechanism of causality has been found to be blockage by mercury and other toxic
metals of the enzymatic processes related to digestion of milk casein and wheat
gluten, resulting in dumping of morphine like substances in the blood of the
majority of children with these conditions(9).
A direct
mechanism involving mercury’s inhibition of cellular enzymatic processes by
binding with the hydroxyl radical(SH) in amino acids
appears to be a major part of the connection to allergic/immune reactive
conditions(15-23,98,4). For example mercury has been found to strongly inhibit
the activity of xanthine oxidase(16) and dipeptyl peptidase (DPP IV) which are required in the
digestion of the wheat protein gluten or the milk protein casein
(4,15,17,19,20,24-26,98,499) - the same protein that is cluster
differentiation antigen 26 (CD26) which helps T lymphocyte activation. CD26 or
DPPIV is a cell surface glycoprotein that is very susceptible to inactivation
by mercury binding to its cysteinyl domain. Mercury and other toxic metals also
inhibit binding of opioid receptor agonists to opioid receptors, while
magnesium stimulates binding to opioid receptors(15).
Beta-casomorphine-7 is a
morphine like compound that results in neural disfunction (24,25,4), as well as
being a direct histamine releaser in humans and inducing skin reactions
(14,21,25c).
A similar
mechanism related to mercury and toxic metals blocking the enzymatic processes
related to digestion of gluten and casein have also been shown to be a
significant factor in causality of children’s type I diabetes. Early introduction
of feeding cow’s milk to infants rather breastfeeding has been shown to be a
significant risk factor in type I diabetes incidence (4,5).
Most infants in recent
years have been found to be exposed to high levels of mercury and toxic metals
through vaccines, mother’s amalgam dental fillings, and other sources(8), with toxic metals documented to block the
enzymatic processes needed to digest casein and gluten. Studies have found high
levels of Ig A antigen specific antibodies for casein, lactalbumin and
beta-lactoglobulin and IgG and IgM for casein in cow’s milk(9,4,etc.). In vitro the bioactive peptide beta-casomorphin 7 (BCM-7) is yielded by the successive
gastrointestinal proteolytic digestion of bovine beta-casein variants A1 and B,
but this was not seen in variant A2 (4,498). In hydrolysed
milk with variant A1 of beta-casein, BCM-7 level is 4-fold higher than in A2
milk. Consumption of the beta-casein A1 variant had a strong correlation to
diabetes incidence (r = +0.726). Even more pronounced was the relation between
beta-casein (A1+B) consumption and diabetes (r = +0.982).(4a)
These latter two cow caseins yield a bioactive peptide beta-casomorphin-7 after
in vitro digestion with intestinal enzymes whereas the common A2 variant or the
corresponding human or goat caseins do not. BCM-7 has also been found to be a factor in autism and
schizophrenia. Mercury and toxic metals have been found to disable the
enzymatic process required to digest casein(9).
A1 is the
most frequent in Holstein-Friesian (0.310–0.660), Ayrshire (0.432–0.720) and
Red (0.710) cattle(4b,498). In contrast, a high
frequency of A2 is observed in Guernsey (0.880–0.970) and Jersey (0.490–0.721)
cattle as well as Asian and African cattle. BCM-7 may play a role in the aetiology of many human diseases including diabetes,
neurological, immune, and cardiovascular(498) .
Epidemiological evidence from New Zealand claims that consumption of
beta-casein A1 is associated with higher national mortality rates from ischaemic heart disease. It seems that the populations that
consume milk containing high levels of beta-casein A2 have a lower incidence of
cardiovascular disease and type 1 diabetes. BCM-7 has also been suggested as a
possible cause of sudden infant death syndrome. In addition, neurological
disorders, such as autism and schizophrenia, apprear
to be associated with milk consumption and a higher level of BCM-7(4b)
Lactose
Intolerance
Lactose
(milk sugar), which is a major component of milk, is a disaccharide sugar made
up of the simple sugars glucose and galactose(3).
Lactase is an enzyme which facilitates digestion of lactose. Over 50% of
non-Caucasians are lactose intolerant, to a significant degree and about 20% of
Caucasians. Infants are most lactose tolerant but lactase activity declines
dramatically over time so that by adulthood to about 5 to 10 % of the level of
infants. Only a relatively small percentage of people retain enough lactase
activity to absorb significant amounts of lactose throughout their adult life
(3). Lactose intolerance results in undigested lactose in the intestines which
often causes gas, bloating, abdominal discomfort, and proliferation of bacteria
in the intestines. In addition to inhibiting the enzymes such as peptidases
required to digest milk casein and whey, chronic mercury exposure in animals
has also been found to inhibit lactase and glucose-6-phosphatase needed to
digest lactose and other polysaccharides (32). Thus
chronic exposure to mercury and toxic metals also increases lactose intolerance
and digestion problems of carbohydrates in general. Digestive problems have
been found to commonly be improved by reducing chronic
mercury and toxic metal exposures.
Lactose
intolerance can also be alleviated to some degree by supplemental enzymes,
using fermented milk products such as yogurt or kefir, or using only small
amounts of milk products spread throughout the day(3).
Rat study findings suggest that mercury-induced changes in RT6+ T
lymphocytes
appear to be related to the development of renal autoimmune insulin-dependent
diabetes mellitus disease in genetically predisposed BN rats(6).
Treatment of a strain of rats that are more prone to diabetes than other
strains with mercury chloride results in significantly higher incidences of
autoimmunity to thyrogobulin and laminin(30).
Mercury has been documented to cause autoimmune conditions like thyroiditis,
MS, Lupus, eczema, etc. and people who replace amalgam fillings commonly
recover or significantly improve from these conditions(369b).
Another study found that
the age or timing of vaccinations containing mercury thimerosal affects the
incidence of diabetes in rats(31). Vanadium, uranium,
and other toxic metals have also been found to damage pancreatic islet cells
and be a factor in diabetes(490).
Until mercury thimerosal
was removed from most vaccines, vaccines were the largest source of mercury in
most infants and young children(8a) and exposures far more than Government
health guidelines were common. Dental amalgam has been documented to be the
largest source of mercury in most who have several amalgam mercury fillings(500) with high typical exposures far more than
Gov’t health guidelines. Dental amalgam has been documented to be the largest
source of mercury in most mothers with several amalgam fillings and the largest
prenatal mercury exposure source to the fetus, as well as significant source of
mercury exposure in infants (501).
Mercury
causes release of inflammatory cytokines such as Tumor Necrosis Factor-alpha(TNFa) and Interleukin-4
which are documented to be factors in the chronic inflammatory conditions, cell
death, and damage to the immune system. (47,65,101) TNFa(tumor necrosis
factor-alpha) is a cytokine that controls a wide range of immune cell response
in mammals, including cell death(apoptosis). Mercury has been shown to induce TNFa, deplete glutathione, and increase glutamate,
dopamine, and calcium related toxicity, causing inflammatory effects and
cellular apoptosis in immune cells(47). Cell signaling
mechanisms like sphingolipids are part of the control mechanism for the TNFa apoptosis mechanism(101).
Glutathione is an amino acid that is a normal cellular mechanism for
controlling apoptosis. When glutathione is depleted, reactive oxidative species
increased, and CNS and cell signaling mechanisms are disrupted by toxic
exposures such as mercury, neuronal cell apoptosis results and cellular damage.
Oxidative stress including methylmercury-induced
oxidative stress causes pancreatic beta-cell apoptosis (programmed cell death)
and dysfunction(491,492,493,499). Thimerosal has
similar toxicity to methyl mercury(8a) and dental amalgam is a significant
source of methyl-mercury in those with amalgam fillings(500).
Both
immune cell type Th1 and Th2 cytokine responses are involved in
autoimmunity(425c). Mercury has been found to affect both Th1 and Th2 cytokines
causing an increase in inflammatory Th2 cytokines(152,181,285,404b)
as well as in Th1 cytokines in some circumstances(6). In the pancreas, the
cells responsible for insulin production can be damaged or destroyed by the
chronic high levels of cytokines, with the potential of inducing type I or II
diabetes - even in otherwise healthy individuals with no other risk factors for
diabetes(502). Mercury inhibits production of insulin
and is a factor in diabetes and hypoglycemia, with significant reductions in
insulin need after replacement of amalgam filings and
normalizing of blood sugar(35,502). Mercury exposure
also increases insulin resistance(505).
Mercury
exposure causes an approximately 1.8-fold increase in glucose transport. This
glucose transport corresponds with an increase in GLUT 1 glucose transporters.
Phosphorylation of p38 kinase increased with mercury exposure. Activation of
p38 and an increase in glucose transport corresponding to an increase in GLUT 1
are indicative the induction of a stress response, which can contribute to the
induction of insulin resistance in adipocytes. While the magnitude of the
action of mercury is modest, its effects were sustained over many days of
exposure and impacted subsequent insulin-mediated glucose transport.
Pre-treatment with HgCl2 decreased insulin-mediated glucose transport 1.3-fold
suggesting that exposure to mercury may contribute to pathologies associated
with glucose homeostasis(10).
Studies
have also found evidence of a connection between low levels of zinc and
childhood-onset diabetes(72) . Zinc is an antagonist
to toxic metals like cadmium and mercury, and adequate levels are required to
balance the adverse effects of these toxic metals on cellular calcium and other
enzymatic processes(28,74). Mercury reduces the
bloods ability to transport essential nutrients such as calcium, magnesium, and
zinc; and has an adverse effect on cellular levels of these essential minerals,
as well as blocking the function of magnesium and zinc in cellular enzymatic functions(43,96,198,338). Part of the toxic effects of
mercury, cadmium, lead, etc. are through their replacing essential minerals
such as zinc at their sites in enzymes, disabling the necessary enzymatic
processes. Other connections between mercury and type 1 diabetes have also been
demonstrated. Mercury inhibits production of insulin and is a factor in
diabetes and hypoglycemia, with significant reductions in insulin need after
replacement of amalgam filings and normalizing of
blood sugar(35).
Dr. J. Bart Classen discovered it would be possible to study the
effect of Hemophilus B immunization on the incidence
of IDDM using data from a large clinical trial
in Finland. Dr. J. Bart Classen and D.C. Classen initiated and funded a
collaboration with Dr. Tuomilehto in Finland. All
children born in Finland between October 1st, 1985 and August 31st, 1987,
approximately 116,000 were randomized to receive 4 doses of the HiB vaccine (PPR-D, Connaught) starting at 3 months of life
or one dose starting at 24 months of life. Classen and Classen calculated the
incidence of IDDM in both groups through age 10 and in an
group which did not receive the HiB vaccine, a cohort
which included all 128,500 children born in Finland in the 24 months prior to
the Hemophilus vaccine study. Immediately following
the completion of these two arms all children born in Finland over a two year period were randomized to receive 3 doses of the
old PPR-D HiB vaccine or 3 doses of a newer HbOC HiB vaccine. The data supports published findings that the immunization
starting after 2 months of life is associated with an increased incidence of
IDDM. Rises in diabetes have been seen in the UK and USA following the
introduction of the hemophilus vaccine (99). Other
studies have found that any vaccine that contains mercury or other substances
that can induce autoimmunity can cause type I or II diabetes and that
vaccinations are a common cause of autoimmunity(100).
III. Insulin Resistance increases incidence of
neurological conditions including Alzheimer’s
Higher insulin and glucose
levels in the blood and deficiency of glucose in brain cells that need it has
been found to lead to neurological problems such as Alzheimer’s (580,581).
Diabetes also impacts memory by increasing the risk blood
vessels will become obstructed, restricting blood flow to the brain.
High blood glucose levels
also impact cognition through formation of sugar-related toxins called advanced
glycation end products (AGEs). AGEs have been found to be a factor in aging,
diabetes, and Alzheimer’s. Glycotoxins are formed
when sugars interact with proteins and lipids, damaging the structure of
proteins and membranes, rendering them less able to carry out their many vital
processes. (581). Studies have shown that AGEs are a key factor in
cross-linking of harmful beta-amyloid plaques in the brain that are implicated
in Alzheimer’s. As previously documented mercury and aluminum exposure increase
insulin resistance and amalgam replacement and detoxification reduce insulin
resistance. Studies have also found that supplementation with chromium improves
memory performance in individuals with mild memory loss, by reducing insulin
resistance and increasing ability of cells to use insulin.
IV. Effect of Persistent
Organic Pollutants(POPs)
Effects of low-level
exposure to some persistent organic pollutants (POPs) was surveyed in a large
population (NHANES) survey(497). There was a strong dose-response relations between serum concentrations
of six selected POPs and the prevalence of diabetes. Diabetes prevalence was
strongly positively associated with lipid-adjusted serum concentrations of all
six POPs. When the participants were classified according to the sum of
category numbers of the six POPs, adjusted odds ratios were strongly
significant for trend. The association was consistent in stratified analyses
and stronger in younger participants, Mexican Americans, and obese individuals.
Systemic detoxification has been found to be effective in reducing the levels
of toxic metals and POPs and to lessen related adverse health effects. There
are many alternatives for accomplishing this that have been demonstrated to
have effectiveness.
V Effective Treatment
or Control of Diabetes
Authorities referenced here
suggest the following measures to prevent diabetes and insulin resistance:
lower toxic exposure levels, control weight and lose weight if overweight,
reduce body fat percentage where excessive, regular exercise, good diet using glycemic
index or glycemic load information, take appropriate supplements in addition to
a good multivitamin/multimineral supplement(see
below), monitor blood glucose level .
For those with several
amalgam dental fillings and diabetes, amalgam replacement and detoxification
commonly results in significant improvement (499,598,etc.).
Reduced consumption of excitoxins like MSG and
aspartame and of high fructose corn syrup and other omega 6 oils along with
increased consumption of magnesium and omega 3 oils such as virgin olive oil
and coconut oil have been found to reduce brain inflammation and improve
inflammatory conditions like insulin resistance, diabetes, depression, etc. (598,599,580,etc.). Drugs such as diuretics, blood pressure medications,
steroids for asthma, arthritis, or allergies, prozac,
and birth control pills that can increase insulin resistance and blood sugar
(1). Drugs used to treat diabetes such as Avandia and Actos have been found to
cause significant increases in cardiovascular disease and heart attacks, as
well as increased risk of hip fracture(581b,FDA,etc.)
Other factors that have
been found to be significantly associated with cardiovascular disease and
diabetes include daily consumption of soda drinks, diet drinks, fried foods, or
a “Western Diet” high in fried foods, refined grains, fast foods, soda, etc.
and low in fruits and vegetables (590,598,580,1). These diet patterns all have
been found to be significantly associated with metabolic syndrome, a cluster
of cardiovascular disease and diabetes risk factors including elevated waist
circumference, high blood pressure, elevated triglycerides, low levels of
high-density lipoprotein (HDL or "good") cholesterol, clogged
arteries, and high fasting glucose levels. The presence of three or more of the
factors increases a person's risk of developing diabetes and cardiovascular
disease. Avoidance of these dietary causes of insulin resistance and metabolic
syndrome along with exercise such as walking, have been found
to be highly effective at preventing or controlling these conditions (1, 598 ,etc.) Some carbohydrates elevate
blood glucose almost instantly -- others much more slowly. The Glycemic
Index (GI) is a measure of how quickly carbohydrates elevate glucose.
A lower number indicates a slower glucose rise -- and better glucose control.
Monitoring and avoiding high glycemic foods along with exercise have been found
to be effective in controlling insulin resistance(1c).
Oily fish,
greens(chlorophyll) and Fenugreek are too foods that have been documented to be
effective in helping to control insulin resistance(1a). Other foods supportive
of controlling blood sugar include foods with high fiber content, whole grain
bread, brown rice, dried beans and peas, fresh vegetables and fruit (1a).
Glycemic Load (GL) takes into account not only the quality of carbohydrate but
also quantity and its effects.
Several studies have
documented that lipoic acid(an antioxidant and
chelator) resulted in improvement in the majority of diabetes cases it was used
for, by improving glucose metabolism, increasing insulin sensitivity, and
reducing nerve damage(including in diabetic neuropathy)(502e) Lipoic acid is
also a mercury chelator, commonly used for that purpose.
Several supplements have
been found to be significantly effective in promoting healthy blood glucose
metabolism. These include Gymnema sylvestre,
bitter melon, N-acetyl-cysteine (NAC), Vitamin C and E, Goat’s Rue, Cinnamon,
Quercetin, Vanadyl sulfate, Bitter melon, PolyGycopleX(PGX) (581,1). Nutrients that have been found effective at
blocking AGEs include: Carnosine, Vitamin B1 and B6, NAC, Guava, chromium, Pycnogenol, B7, magnesium, and Yerbamate
(581).
Several types of organ damage can be caused by diabetes/insulin
resistance. Diabetic neuropathies are one such. Supplements documented to help
with such neuropathies include lipoic acid, Vit B6
and B12, Folic Acid, Fish oil, and Huckleberry leaf tea(1a). Diabetes can also
be a factor in vision problems such as cataracts and diabetic retinopathy.
Supplements found to help with these conditions include Taurine, bilberry,
lycopene (1a). CAN-C drops (N-acytyl-Carnosine)
have been found to be effective at reducing or
reversing damage cataracts or other vision problems (582). Acetyl-L carnitine(ALC) reduces effects and pain of diabetic
neuropathy(581b).
Supplements found to help with diabetic related kidney damage
include Taurine, melatonin, and cranberry proanthocyanidins
(1a). Supplements found to help with diabetic related cardiovascular conditions
include L-Arginine, L-Carnitine, and Hawthorne which can be found in hawthorne tea or supplements (1a).
Chromium has been found to aid insulin transportation into cells.
Cinnamon has been found to lower blood sugar levels. Lipoic acid has been found
to improve glucose balance for people with type II diabetes. CoQ10 has
cardiovascular benefits and prevents high blood pressure in people with blood
glucose control problems. (581b)
VI. Diabetes Tests:
Fasting Glucose Test(optimal: 76-81 mg/dL, normal: 82-85 mg/dL prediabetic: 86-125, diabetic: >126 mg/dL), Glucose test insulin level: (optimal: =<
7mcU/ml, prediabetic: 8-25 mcU/ml, diabetic: >25 mcU/ml); Fasting Glucose Tolerance Test with glucose and
insulin levels: (normal: glucose < 140 mg/dL
& insulin level <55 mcU/ml, prediabetic: blood
glucose level of 140 to 159 mg/dL or insulin level of
56-90 mg/dl, dangerous: glucose> 160 mg/dL or
insulin >90 mcU/ml); Hemoglobin A1C(HbAiC) (measures damage to blood proteins by free
radicals created) (normal: 4.5-4.9%, prediabetic: 5-6.9%, Diabetic:
>7%)
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_______________________________________
Dr. J. Bart Classen
discovered it would be possible to study the effect of Hemophilus
B immunization on the incidence of IDDM using
data from a large clinical trial in Finland. Dr. J. Bart Classen
and D.C. Classen initiated and funded a collaboration with Dr. Tuomilehto in Finland. All children born in Finland between
October 1st, 1985 and August 31st, 1987, approximately 116,000 were randomized
to receive 4 doses of the HiB vaccine (PPR-D,
Connaught) starting at 3 months of life or one dose starting at 24 months of
life. Classen and Classen calculated the incidence of IDDM in both groups
through age 10 and in an group which did not receive
the HiB vaccine, a cohort which included all 128,500
children born in Finland in the 24 months prior to the Hemophilus
vaccine study. Immediately following the completion of these two arms all
children born in Finland over a two year period were
randomized to receive 3 doses of the old PPR-D HiB
vaccine or 3 doses of a newer HbOC HiB vaccine. The data supports published findings that the immunization
starting after 2 months of life is associated with an increased incidence of
IDDM. Rises in diabetes have been seen in the UK and USA following the
introduction of the hemophilus vaccine (see HIB)
Dr. J. Bart Classen, Hemophilus
Vaccine Study in Finland Proves a Causal Relationship Between Vaccines and
Diabetes, Autoimmunity 35:247-253,2002,
http://www.vaccines.net/hemophil.htm
Congressional
Testimony by Dr. J. Bart Classen regarding the link between vaccines and diabetes in
the military. http://www.whale.to/vaccines/classen3.html
Dr.
J. Bart Classen, Hemophilus Meningitis Vaccine Proven to Cause Diabetes in
Clinical Trial of Over 100,000 Children, Autoimmunity,
Vol 39, July 2004
The
study followed over 100,000 children which had been randomized in a large
clinical trial to receive 1 or 4 doses of the hemophilus
vaccine and over
100,000 unvaccinated children. After 7 years the group receiving 4 doses
of
the vaccine had a statistically significant 26% elevated rate of diabetes, or
an extra 54 cases/100,000 children, compared to children who did not receive
the vaccine. By contrast immunization against hemophilus
is expected to
prevent only 7 deaths and 7 to 26 cases of permanent disability per 100,000
children immunized. The study showed that almost all of the extra cases
of
diabetes caused by the vaccine occurred between 3-4 years after vaccination.
Furthermore the paper provides new data proving the
vaccine causes diabetes in
mice and reviews data from 3 smaller human studies, which all had similar
results to the current study, but were too small to reach statistical
significance.
"Our results conclusively prove there is a causal relationship between
immunization schedules and diabetes. We believe immunization schedules
can be
made safer," stated Dr. Bart Classen.
Classen
JB. The diabetes epidemic and the hepatitis B vaccines. N Z Med J.
1996 Sep 27;109(1030):366.
Classen,
JB, MD, "The Timing of Immunization Affects The
Development of Diabetes in Rodents", Autoimmunity, 1996, 24:137-145.
Classen,
J.B. Congressional Testimony on the connection between vaccines and diabetes, http://www.whale.to/v/classen.html